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Immune Cell Biology Program, Naval Medical Research Institute | 論文
- CD28 co-stimulatory signals induced IL-2 receptor expression on antigen-stimulated virgin T cells by an IL-2- independent mechanism.
- The role of B7-1a molecule, an alternatively spliced form of murine B7-1(CD80), on T cell activation.
- CD28 co-stimulatory signals induce IL-2 receptor expression on antigen-stimulated virgin T cells by an IL-2-independent mechanism
- The lgV domain of human B7-2 (CD86) is sufficient to co-stimulate T lymphocytes and induce cytokine secretion
- The role of B7-CD28 co-stimulation in tumor rejection
- Both CD28 ligands CD80(B7-1) and CD86(B7-2) activate phosphatidylinositol 3-kinase, and wortmannin reveals heterogeneity in the regulation of T cell IL-2 secretion
- CD28 ligands CD80(B7-1) and CD86(B7-2) induce long-term autocrine growth of CD4+T cells and induce similar patterns of cytokine secretion in vitro
- Primary porcine endothelial cells express membrane-bound B7-2 (CD86) and a soluble factor that co-stimulate cyclosporin A-resistant and CD28-dependent human T cell proliferation