スポンサーリンク
First Department of Pathology, Nagoya city University Medical School | 論文
- Resveratrol Inhibits Angiogenic Response of Cultured Endothelial F-2 Cells to Vascular Endothelial Growth Factor, but Not to Basic Fibroblast Growth Factor
- SPONTANEOUS TUMORS IN F344/DuCrj RATS
- Establishment of an in vivo Highly Metastatic Rat Hepatocellular Carcinoma Model
- Clinicopathological analysis on cancers of autopsy cases in a geriatric hospital
- Histopathological Analysis of Invasive Bladder Carcinomas Induced by 3,2'-Dimethyl-4-aminobiphenyl in Hamsters
- HISTOPATHOLOGICAL EVALUATION OF SPONTANEOUS HEPATOCELLULAR TUMORS IN B6C3F_1 HYBRID MICE
- Suppressive Effects of Dietary Genistin and Daidzin on Rat Prostate Carcinogenesis
- Development of Androgen-independent Carcinomas from Androgen-dependent Preneoplastic Lesions in the Male Accessory Sex Organs of Rats Treated with 3,2'-Dimethyl-4-aminobiphenyl and Testosterone Propionate
- Effects of Verapamil on PhIP-induced Tumorigenesis in F344 Rats
- Medium-term Rat Liver Bioassay for Rapid Detection of Hepatocarcinogenic Substances
- SYNERGISM IN HEPATOCARCINOGENESIS INDUCED BY HETEROCYCLIC AMINES IN RATS
- ENHANCING EFFECTS OF HIGH-FAT DIET ON 3, 2'-DIMETHYL-4-AMINOBIPHENYL URINARY BLADDER CARCINOGENESIS IN THE HAMSTER
- ヘテロサイクリックアミンの複合によるラット肝発癌の促進
- WEAK SYNERGISTIC EFFECT OF URACIL AND N-BUTYL-N-(4-HYDROXYBUTYL)NITROSAMINE ON URINE COMPOSITION, EPITHELIAL MORPHOLOGY, AND DNA SYNTHESIS IN THE URINARY BLADDER OF SYRIAN GOLDEN HAMSTER
- EFFECTS OF HEMODIALYSIS ON AMYLOID DEPOSITION
- Establishment of Transplantable Rat Prostate Carcinomas from Primary Lesions Induced by 3,2'-Dimethyl-4-aminobiphenyl and Testosterone
- Modification of the Carcinogenic Potency of Quinoline, a Hepatocarcinogen, by Fluorine Atom Substitution : Evaluation of Carcinogenicity by a Medium-Term Assay
- Growth Characteristics in the Initial Stage of Micrometastasis Formation by Bacterial LacZ Gene-tagged Rat Prostatic Adenocarcinoma Cells
- P-51(O-1) ラット乳腺においてPhIPにより誘発されたlacI遺伝子の突然変異の解析
- CHARACTERISTICS OF FORESTOMACH CARCINOGENESIS BY NON-GENOTOXIC PHENOLIC COMPOUNDS