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Department of Cardiovascular Medicine, The University of Tokyo Graduate School of Medicine | 論文
- Acyclic Retinoid Prevents Cardiovascular Remodeling by Regulated KLF5(Cardiovascular Pharmacology, Basic/Clinical 1 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- The Cardiovascular Transcription Factor Kruppel-like Factor 5 is Negatively Regulated by the Deacetylase HDAC1 through Direct Interaction(Molecular Biology, Vascular 1 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- PE-343 Novel Regulatory Pathway of PDGF-A Chain as Mediated by Transcription Factor KLF5 through Cooperative Activation with NF-κB and Phorbol Ester(Molecular Biology, Vascular 3 (H) : PE59)(Poster Session (English))
- OE-273 Acyclic Retinoid Regulates KLF5 And Inhibits Cardiovascular Remodeling(Cardiovascular Pharmacology, Basic/Clinical 1 (H) : OE34)(Oral Presentation (English))
- OE-102 Positive and negative regulation of the cardiovascular transcription factor KLF5 by p300 and SET through interaction and acetylation(Molecular Biology, Vascular 1 (H) : OE12)(Oral Presentation (English))
- OE-100 The Cardiovascular Remodeling Transcription Factor KLF5 Is Negatively Regulated by the Histone Deacetylase HDAC1 through Direct Interaction(Molecular Biology, Vascular 1 (H) : OE12)(Oral Presentation (English))
- Proteomic Identification of a Transcriptional Represser of the Transcription Factor KLF5/BTEB2
- KLF5 not only Synergistically Activates PDGF-A Gene with NFκ B but also Accelerates SMC Proliferation
- DPE-071 Retinoic Acid Receptor Antagonist Ameliorates Pressure Overload-Induced Cardiac Hypertrophy and Remodeling(DPE12,Cardiomyopathy/Hypertrophy (Basic) (M),Digital Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation
- FRS-093 Significance of the Transcription Factor KLF5 in Myocardial Hypertrophy in Response to Pressure Overload(FRS19,Genetic Abnormalities in Cardiac Hypertrophy and Cardiomyopathy (M),Featured Research Session (English),The 73rd Annual Scientific Meeti
- PJ-412 S100A8 and S100A9 are novel proinflammatory signal mediators in cardiovascular and metabolic disease(Atherosclerosis, basic(07)(IHD),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-256 Significance of the Transcription Factor KLF5 in Myocardial Hypertrophy in Response to Pressure Overload(Cardiac hypertrophy, basic/clinical(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- Elucidation of Sulfur Behavior on Alumina-Supported Ruthenium Sulfide-Cesium Catalysts Using Radioactive ^S as a Tracer
- Tri-axial Grain Orientation of Y_2Ba_4Cu_7O_y Achieved by the Magneto-science Method
- Rare-Earth-Dependent Magnetic Anisotropy in REBa_2Cu_3O_y
- Stress-induced second harmonic generation in silica glass
- Luminescent properties of amorphous Al_2O_3 prepared by sol-gel method
- Hydrodesulfurization of Dibenzothiophene Catalyzed by Alumina-Supported Ruthenium Carbonyl-Alkali Metal Hydroxide Systems
- STUDIES ON THE INCREASED POTASSIUM LEVEL OF PLASMA PRODUCED BY HYPER-TONIC SOLUTIONS OF LOW POTENT SUBSTANCES INTRAPERITONEALLY ADMINISTERED TO RATS
- Deep Hydrodesulfurization of Alkyl-Substituted Dibenzothiophenes in Light Oil
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