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Department of Cardiovascular Medicine, Nara Medical University | 論文
- OJ-120 New Thrombolytic Technique with Ultrasound in Acute Coronary Syndrome(Thromboembolism/Antithrombotic therapy/Thrombolysis-2 (H) OJ20,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- Plasma level of CD40L correlates with coronary circulating matrix metalloprotainase-9 in patients with coronary artery disease
- Relationship between plasma cardiotrophin-1 and left ventricular remodeling in patients with first acute myocardial infarction
- Possible involvement of Atypical Protein Kinase C in Aldosterone-induced-Angiotensin-Converting-Enzyme Gene Expression in Cultured Neonatal Rat Cardiocytes
- Peroxisome proliferation-activated receptor agonists inhibit matrix metalloproteinase activity of human vascular endothelial cells
- β2 adrenergic receptor antagonism inhibits matrix metalloproteinase-9 activation in human endothelial cells
- Increased risk of coronary spasm due to the combination of-786T to C mutation in eNOS gene and smoking
- Detection of CYP17 gene expression and the regulatory mechanisms in human vascular smooth muscle cells
- Proopiomelanocortin (POMC) gene expression in the human autopsy heart
- Endogenous production of nitric oxide correlates to ventilatory response to exercise and parasympathetic activity in patients with myocardial infarction
- Dual Gradient-echo In-phase and Opposed-phase Magnetic Resonance Imaging to Evaluate Lipomatous Metaplasia in Patients with Old Myocardial Infarction
- Osteoclast differentiation in ectopic bone formation induced by recombinant human bone morphogenetic protein 2 (rhBMP-2)
- Aldosterone is Produced From the Ventricles in Patients With Essential Hypertension
- Adrenocorticotropin (ACTH) Production in the Hypertensive Human Heart
- Evidence for Cardiac Myocyte Regeneration in Injured Myocardium by Bone Marrow Derived Cells
- Overxepression of brain natriuretic peptide mediates cardiac matrix metalloproteinase induction and induces cardiac rupture after acute myocardial infarction in mice
- Inhibition of histone deacetylase selectively induces expression of cardiac embryonic genes that contain neuron-restrictive silencer element in cultured ventricular myocytes
- Disruption of natriuretic peptides receptor A leads to higher mortality and exaggerated left ventricular remodeling after myocardial infarction in mice
- Primary Seminoma in the Middle Mediastinum
- Subacute Cor Pulmonale due to Tumor Embolism