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Department of Cardiology, Gifu University School of Medicine | 論文
- PJ-474 Detection of Coronary Atherosclerotic Plaques by Multi-detector Row Computed Tomography : Comparison to Integrated Backscatter Intravascular Ultrasound(CT/DSA-10, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- Granulocyte Colony-Stimulating Factor : A Noninvasive Regeneration Therapy for Treating Atherosclerotic Peripheral Artery Disease
- PJ-163 Oxytocin Treatment after Myocardial Infarction Promotes Cardiac Repair and Neovascularization through Activation of Multiple Pro-survival Signals(PJ028,Cardiovascular Pharmacology (Basic), Cost-health care system/DPC/Laws (H),Poster Session (Japane
- OE-070 Post-Infarct Treatment with an Erythropoietin-Gelatin Hydrogel Patchlmproves Cardiac Function by Activating Prosurvival Signaling and exerting Antifibrotic and Angiogenic Effects(Genetics/Genetically engineered models/Gene therapy(M),Oral Presentat
- OJ-254 Erythropoietin Drug Delivery System protects the heart against myocardial infarction through activation of Akt and angiogenesis(Growth factor/Cytokine-2(H/M), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- OE-128 Pioglitazone, an Anti-Diabetic Drug, Reduces Myocardial Infarct Size via Stimulating Peroxisome Proliferator Activated Receptor-γ (PPARγ) in Rabbits Model of Myocardial Infarction(Myocardial ischemia/reperfusion, basic/clinical-1, The 71st Annual S
- NRSF Is Involved in Molecular Pathways for Sudden Death Associated with Heart Failure
- PJ-795 Narrow QRS Duration Predicts Improvement of Left Ventricular Systolic Function by Beta-blockers in Chronic Heart Failure(PJ133,Heart Failure (Treatment) 4 (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation
- OJ-314 Drug Delivery System Using Erythropoietin Gelatin Hydrogel Sheet Decreases Myocardial Infarct Size and Improves Left Ventricular Function and Remodeling(Growth factor/Cytokine-1 (H) OJ53,Oral Presentation (Japanese),The 70th Anniversary Annual Scie
- PJ-225 Tropomyosin as One of the Earliest Target of Apoptotic Proteolysis Facilitating Cytoskeletal Catastrophe in Cardiac Myocytes(Heart Failure, Basic 2 (M) : PJ38)(Poster Session (Japanese))
- PE-385 G-CSF Upregulates Myocardial Akt and Improves Function of the Hearts with Established Heart Failure due to Large Old Myocardial Infarction(Old Myocardial Infarction/Remodeling 3 (IHD) : PE66)(Poster Session (English))
- OJ-508 Autophagy, but Not Apoptosis, May be the Mode of Myocyte Death in Hearts of the Cardiomyopathic UM-X7.1 Strain Hamster(Cardiomyopathy, Basic (M) : OJ62)(Oral Presentation (Japanese))
- PJ-440 Ginsenoside Rg1 Reduces Myocardial Infarct Size via Activation of Protein Kinase C and Production of Nitric Oxide in Rabbits(Acute myocardial infarction, basic-4 (IHD) PJ74,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of
- PJ-343 Post-infarction Treatment with Ginsenoside Rg1 Improves Cardiac Function and Left Ventricular Remodeling Through Enhancement of Intrinsic G-CSF(Cardiovascular pharmacology, basic/clinical-4 (H) PJ58,Poster Session (Japanese),The 70th Anniversary An
- PE-078 Pravastatin Reduces Myocardial Infarct Size Through Nitric Oxide Production, Protein Kinase C Activation, Mitochondrial KATP Channel Opening and Decreasing Oxyradicals(Cardiovascular pharmacology, basic/clinical-3 (H) PE13,Poster Session (English),
- Acarbose, an α-glucosidase Inhibitor, Reduces Myocardial Infarct Size in Rabbits Involving the Opening of Mitochondrial KATP Channels(Diabetes/Obesity 8 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Accelerated Post-infarct Neovascularization and Tissue Repair by Chemotherapy-combined G-CSF May Be Related to Peripheral CD34^+ Cells Increased via CXCR4-SDF-1 Axis(Apoptosis/Necrosis/Regeneration 1 (M), The 69th Annual Scientific Meeting of the Japanese
- HGF Gene Transfer is Protective Against Ischemia-Reperfusion Injury via Enhancing Angiogenesis and Bcl-2 Expression and Decreasing Hydroxyl Radicals(The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-584 Nicorandil Reduces the Production of Hydroxyl Radicals During Ischemia and Reperfusion Through Opening the Mitochondrial KATP Channels in Rabbit Hearts(Cardiovascular Pharmacology, Basic/Clinical 5 (H) : PJ98)(Poster Session (Japanese))
- PJ-377 Healing Process Acceleration May be Important as a Mechanism of Improvement of Cardiac Function and Remodeling by Post-infarct G-CSF Treatment(Apoptosis/Necrosis/Regeneration 1 (M) : PJ63)(Poster Session (Japanese))
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