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Department of Cardiology, Gifu University Graduate School of Medicine | 論文
- OJ-508 Autophagy, but Not Apoptosis, May be the Mode of Myocyte Death in Hearts of the Cardiomyopathic UM-X7.1 Strain Hamster(Cardiomyopathy, Basic (M) : OJ62)(Oral Presentation (Japanese))
- Celiprolol, a Selective β_1-Blocker, Reduces the Infarct Size Through Production of Nitric Oxide in a Rabbit Model of Myocardial Infarction
- Nicorandil Improves Post-Ischemic Myocardial Dysfunction in Association With Opening the Mitochondrial KATP Channels and Decreasing Hydroxyl Radicals in Isolated Rat Hearts
- PJ-440 Ginsenoside Rg1 Reduces Myocardial Infarct Size via Activation of Protein Kinase C and Production of Nitric Oxide in Rabbits(Acute myocardial infarction, basic-4 (IHD) PJ74,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of
- PJ-343 Post-infarction Treatment with Ginsenoside Rg1 Improves Cardiac Function and Left Ventricular Remodeling Through Enhancement of Intrinsic G-CSF(Cardiovascular pharmacology, basic/clinical-4 (H) PJ58,Poster Session (Japanese),The 70th Anniversary An
- PE-078 Pravastatin Reduces Myocardial Infarct Size Through Nitric Oxide Production, Protein Kinase C Activation, Mitochondrial KATP Channel Opening and Decreasing Oxyradicals(Cardiovascular pharmacology, basic/clinical-3 (H) PE13,Poster Session (English),
- Acarbose, an α-glucosidase Inhibitor, Reduces Myocardial Infarct Size in Rabbits Involving the Opening of Mitochondrial KATP Channels(Diabetes/Obesity 8 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Accelerated Post-infarct Neovascularization and Tissue Repair by Chemotherapy-combined G-CSF May Be Related to Peripheral CD34^+ Cells Increased via CXCR4-SDF-1 Axis(Apoptosis/Necrosis/Regeneration 1 (M), The 69th Annual Scientific Meeting of the Japanese
- HGF Gene Transfer is Protective Against Ischemia-Reperfusion Injury via Enhancing Angiogenesis and Bcl-2 Expression and Decreasing Hydroxyl Radicals(The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- PJ-584 Nicorandil Reduces the Production of Hydroxyl Radicals During Ischemia and Reperfusion Through Opening the Mitochondrial KATP Channels in Rabbit Hearts(Cardiovascular Pharmacology, Basic/Clinical 5 (H) : PJ98)(Poster Session (Japanese))
- PJ-377 Healing Process Acceleration May be Important as a Mechanism of Improvement of Cardiac Function and Remodeling by Post-infarct G-CSF Treatment(Apoptosis/Necrosis/Regeneration 1 (M) : PJ63)(Poster Session (Japanese))
- PE-270 Intravenous Transplantation of Autologous Bone Marrow-Derived Mononuclear Cells Improves Cardiac Function via Downregulation of TGF-beta and TNF-alpha in Ischemia-Reperfusion Hearts.(Transplantation 1 (M) : PE46)(Poster Session (English))
- OE-110 G-CSF after Transient Myelosuppression Improves Cardiac Function via More Myocardial Regeneration and Vasculogenesis than G-CSF Alone in Ischemia-Reperfusion Hearts(Heart Failure, Basic 1 (M) : OE13)(Oral Presentation (English))
- Tendai-Uyaku Improves Post-Ischemic Left Ventricular Dysfunction through Inhibiting Production of Hydroxy Radicals and Opening the Mitochondrial KATP Channels
- Nicorandil Has a Free Radical Scavenging Effect through Opening the Mitochondrial KATP Channels during Ischemia and Reperfusion in Rabbits
- Miglitol Reduces Myocardial Infarct Size via Scavenging Hydroxyl Radicals, Enhancing Bel-XL Expression and Reducing Bax Expression in Rabbits
- HGF Gene Transfer Reduces Myocardial Infarct Size through Decreasing Hydroxyl Radicals Production and Enhancing Expression of BcI-2 in Rabbits
- G-CSF Treatment after Myocardial Infarction Improves Left Ventricular Function and Remodeling Even at 3 Months after Infarction
- Large Mass Scale by Strong Gauge Dynamics with Infrared Fixed Point(Particles and Fields)
- Large Mass Scale by Strong Gauge Dynamics with Infrared Fixed Point
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