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Department Of Phamacology University Of Occupational And Environmental Health | 論文
- Inhibition by Selenium Compounds of Catecholamine Secretion Due to Inhibition of Ca^ Influx in Cultured Bovine Adrenal Chromaffin Cells
- Effect of cAMP-Dependent Protein Kinase on Catecholamine Secretion from Bovine Adrenal Chromaffin Cells
- FRS-084 Spontaneous Myocardial Infarction in Mice Lacking All Nitric Oxide Synthase Isoforms(Molecular Biology in Cardiovascular Diseases (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- FRS-021 Long-Term Treatment with an Angiotensin II Type 1 Receptor Blocker Ameliorates Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthases(Diabetes/Obesity (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
- Development of Genetically Engineered Mice Lacking All Three Nitric Oxide Synthases
- cDNA Cloning of Inducer Reactive Protein in Adult T-Cell Leukemia Cells by Applying the Fuzzy Set Cloning Theory for Sequencing Defuzzification
- Enhanced Expression of CX3CR1 in Patients with Coronary Restenosis(Restenosis after Angioplasty, Basic/Clinical 2 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Atorvastatin Upregulates Vascular Neuronal Nitric Oxide Synthase Expression Through the Akt/NF-κB Pathway(Endothelium/NO 4 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Vascular Neuronal Nitric Oxide Synthase Expression is Upregulated in Response to Platelet-Derived Growth Factor Through the MEK/ERK Pathway(Endothelium/NO 4 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Neuronal Nitric Oxide Synthase Is Subject to Expressional Regulation by Inflammatory Stimuli in Vascular Smooth Muscle Cells in Rats
- Complete Disruption of All Nitric Oxide Synthase Genes Causes Markedly Accelerated Renal Lesion Formation Following Unilateral Ureteral Obstruction in Mice In Vivo
- OJ-163 Genetic Disruption of All Nitric Oxide Synthase Isoforms Abolishes EDHF-Mediated Responses in Mice(Endothelium-2 (H) OJ28,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- Functional Consequences of Substitution of the Disulfide-Bonded Segment, Cys127-Cys150, Located in the Extracellular Domain of the Na, K-ATPase β Subunit: Arg148 Is Essential for the Functional Expression of Na, K-ATPase
- Spontaneous Development of Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthases
- Inhibitoion of LDL Oxidation by Angiotensin Receptor Blockers in Hypertensive Patients (Cardiovascular Pharmacology, Basic/Clinical 2 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- ACTIVATION BY PHOSPHOLIPASE A2 OF THE CATECHOLAMINE RELEASE FROM ISOLATED CHROMAFFIN GRANULES
- Complete Disruption of All Nitric Oxide Synthase Genes Causes Markedly Accelerated Renal Lesion Formation Following Unilateral Ureteral Obstruction in Mice In Vivo
- Inhibition by Pregnenolone Sulphate, a Metabolite of the Neurosteroid Pregnenolone, of Voltage-Gated Sodium Channels Expressed in Xenopus Oocytes
- Binding of titanium dioxide nanoparticles to lactate dehydrogenase
- Inhibition by Pregnenolone Sulphate, a Metabolite of the Neurosteroid Pregnenolone, of Voltage-Gated Sodium Channels Expressed in Xenopus Oocytes
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