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Department Of Neuropsychiatry Kagawa Medical School | 論文
- A Novel Diagnostic Method for Human Immunodeficiency Virus Type-1 in Plasma by Near-Infrared Spectroscopy
- Interleukin-4 Up-Regulates T-Tropic Human Immunodeficiency Virus Type 1 Transcription in Primary CD4^+ CD38^+ T-Lymphocyte Subset
- Human Immunodeficiency Virus Type 1 Subtype C Exhibits Higher Transactivation Actvity of Tat than Subtypes B and E
- Exposure of Normal Monocyte - Drived Dendritic Cells to Human Immunodeficiency Virus Type - 1 Particles Leads to the Induction of Apoptosis in Co - Cultured CD4^+ as Well as CD8^+ T Cells
- Protease-defective, gp120-containing human immunodeficiency virus type 1 particles induce apoptosis more efficiently than does wild-type virus or recombinant gp120 protein in healthy donor-derived peripheral blood T cells.
- Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25 killer cells in a small subset, leading to induction of apoptosis in bystander cells
- Possible Correlation between Borna Disease Virus Infection and Japanese Patients with Chronic Fatigue Syndrom
- Demonstration of Borna disease virus RNA in peripheral blood mononuclear cells from healthy horses in Japan.
- Prevalence of Borna disease virus RNA in peripheral blood mononuclear cells from blood donors.
- Viral activation from latency during retrodifferentiation of U937 cells exposed to phorbol ester followed by infection with human immunodeficiency virus type 1.
- High susceptibility of U937-derived subclone to infection with human immunodeficiency virus type 1 is correlated with virus-induced celldifferentiation and superoxide generation.
- Stimulation of human immunodeficiency virus type 1 infected cells with superoxide enhances the chemotactic motile response of CD4+ human cells: implication for virus transmission by cell-to-cell interaction.
- Changes of Bcl-2 and Bax mRNA Expressions in the Ethanol-Treated Mouse Brain
- Borna Disease Virus Infection in Two Family Clusters of Patients with Chronic Fatigue Syndrome
- Amplification of a Full-Length Borna Disease Virus(BDV) cDNA from Total RNA of Cells Persistently Infected with BDV
- HEREDITARY DRPLA FAMILY WITH A MARKEDLY INCREASED NUMBER OF CAG REPEATS IN MATERNAL TRANSMISSION
- Maintenance of high virus load even after seroconversion in newborn cats acutely infected with feline immunodeficiency virus.
- Superinfection of a defective human immunodeficiency virus type 1 provirus-carrying T cell clone with vif or vpu mutants gives cytopathic virus particles by homologous recombination.
- Demonstration of human Borna disease virus RNA in human peripheral blood mononuclear cells.
- Naturally occurring accessory gene mutations lead to persistent human immunodeficiency virus type 1 infection of CD4-positive T cells.