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Department Of Cardiovascular Medicine Nano-bioengineering Education Program Graduate School Of Medic | 論文
- Kruppel-Like Zinc-Finger Transcription Factor KLF5/BTEB2 ls a Target for Angiotensin II Signaling and an Essential Regulator of Cardiovascular Remodeling.
- Obese adipose tissue remodeling, malfunctioning, and chronic inflammation visualized by in vivo molecular imaging
- PJ-178 Free Fatty Acid, Palmitate, Induces Metabolic and Vascular Lipotoxicity via the IRE1 Arm of ER Stress Response Pathways(PJ030,Metabolic Syndrome 1 (H),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
- 4 Bone marrow-derived cells contribute to vascular wall inflammation without differentiating into smooth muscle cells(Young Investigator's Award Finalists Lectures (YIA),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
- 0965 HYPOXIA INDUCES VASCULAR ENDOTHELIAL GROWTH FACTOR IN CULTURED HUMAN ENDOTHELIAL CELLS
- PE-343 Novel Regulatory Pathway of PDGF-A Chain as Mediated by Transcription Factor KLF5 through Cooperative Activation with NF-κB and Phorbol Ester(Molecular Biology, Vascular 3 (H) : PE59)(Poster Session (English))
- Proteomic Identification of a Transcriptional Represser of the Transcription Factor KLF5/BTEB2
- KLF5 not only Synergistically Activates PDGF-A Gene with NFκ B but also Accelerates SMC Proliferation
- Angiotensin II downregulates renal expression of anti-aging gene, kiotho, and exogenous klotho gene transfer ameliorates angiotensin II-induced renal damage
- DPE-071 Retinoic Acid Receptor Antagonist Ameliorates Pressure Overload-Induced Cardiac Hypertrophy and Remodeling(DPE12,Cardiomyopathy/Hypertrophy (Basic) (M),Digital Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation
- OE-057 Saturated Fatty Acid, Palmitate, Promotes Smooth Muscle Phenotypic Modulation and Exacerbates Neointima Formation(OE10,Atherosclerosis, Basic 1 (IHD),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society
- FRS-104 KLF5 Controls Tissue Remodeling in Cardiovascular and Kidney Diseases via S100 Proteins(FRS21,New Insights into Chronic Kidney Disease and Renal Circulation (H),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japanese
- FRS-093 Significance of the Transcription Factor KLF5 in Myocardial Hypertrophy in Response to Pressure Overload(FRS19,Genetic Abnormalities in Cardiac Hypertrophy and Cardiomyopathy (M),Featured Research Session (English),The 73rd Annual Scientific Meeti
- FRS-079 Rad51 Plays a Pivotal Role in Adipose Tissue Obesity and Systemic Insulin Resistance(FRS16,Basic Mechanisms of Lipid Disorders and Metabolic Syndrome (H),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japanese Circul
- PJ-412 S100A8 and S100A9 are novel proinflammatory signal mediators in cardiovascular and metabolic disease(Atherosclerosis, basic(07)(IHD),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- PE-183 KLF5 Plays an Important Role in Endoplasmic Reticulum Stress Response in both Cardiovascular and Metabolic Systems(Diabetes/Obesity/Metabolic syndrome(05)(H),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Soc
- PE-030 Endoplasmic Reticulum Stress Signaling Modulates Smooth Muscle Phenotypes(Atherosclerosis, basic(03)(IHD),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-256 Significance of the Transcription Factor KLF5 in Myocardial Hypertrophy in Response to Pressure Overload(Cardiac hypertrophy, basic/clinical(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- OE-235 Identification of Metabolic Syndrome Related-unknown Key Molecule With Using siRNA Library(Metabolism/Biochemistry/Energetics(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
- FRS-093 KLF5 controls fibrosis in cardiovascular and kidney diseases via S100 protein expression(Renal Dysfunction and Cardiovascular Disease(H),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
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