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Department Of Cardiology Osaka University School Of Medicine | 論文
- FRS-008 Clinical Impact of Smoking Cessation on Cardiac Events in Survived Patients with Acute Myocardial Infarction(Strategies to Acute Myocardial Infarction-2 (IHD) FRS2,Featured Research Session,The 70th Anniversary Annual Scientific Meeting of the Jap
- FRS-007 Clinical Impact of Metabolic Syndrome on Cardiac Events in Survived Patients with Acute Myocardial Infarction(Strategies to Acute Myocardial Infarction-2 (IHD) FRS2,Featured Research Session,The 70th Anniversary Annual Scientific Meeting of the Ja
- 1 Mortality of Patients with Poor Left Ventricular Function after Myocardial Infarction in Japan : Determinants of Sudden Cardiac Death(Prognosis and treatment strategies for the Japanese patients with left ventricular systolic dysfunction,Controversy 1 (
- Impact of 87 Gene Polymorphisms on Mortality and Recurrent Events After Myocardial Infarction(Acute Myocardial Infarction, Clinical (Pathophysiology) 6 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- -P129- SUSTAINED MYOCARDIAL ISCHEMIA AFTER PERCUTANEOUS TRANSLUMINAL CORONARY ANGIOPLASTY
- -P127- EFFECT OF REVASCULARIZATION ON AREA AND TRANSMURALITY OF INFARCTION IN RELATION TO ELAPSED TIME
- -300- COMPARISON OF PRESSURE HALF-TIME METHOD AND CONTINUITY EQUATION METHOD IN QUANTITATING MITRAL VALVE AREA : Echo Cardiography : FREE COMMUNICATIONS(II) : PROCEEDINGS OF THE 51th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY
- Presenilin 2 Regurates Excitation-contraction Coupling in Hearts(Excitation-contraction Coupling/Ion Channel 1 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- OJ-525 Targeted Deletion of Apoptosis Signal-regulating Kinase 1 Attenuates Left Ventricular Remodeling(Molecular Biology, Myocardium 2 (M) : OJ64)(Oral Presentation (Japanese))
- OJ-524 The Protective Role of STAT6 in Response to Pressure Overload in Vivo(Molecular Biology, Myocardium 2 (M) : OJ64)(Oral Presentation (Japanese))
- OJ-023 Cardiac-specific Over-expression of Sarcolipin Inhibits the Sarcoplasmic Reticulum Ca^ ATPase (SERCA2a) and Reduces the Cardiac Function in Mice(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
- OJ-020 Presenilin 2 Directly Interacts with Cardiac Ryanodine Receptor(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
- OE-367 The Ca^/calmodulin-dependent Protein Kinase II Induces Cardiac Myocyte Hypertrophy through the Activation of Apoptosis Signal-regulating Kinase 1 and Nuclear Factor-κB(Cardiac Hypertrophy, Basic and Clinical 1 (M) : OE46)(Oral Presentation (Eng
- Proline-Rich Tyrosine Kinase 2, Pyk2, Mediates Endothelin-1-Induced Cardiomyocyte Hypertrophy
- Cardiac-Specific Overexpression of a High Ca^ Affinity Mutant of Cardiac Ca^ ATPase Attenuates in Vivo Pressure Overload Cardiac Hypertrophy
- The Small G Protein Rac1 Induce Cardiac Myocyte Hypertrophy through the Activation of Apoptosis Signal-Regulating Kinase1 and Nuclear Factor-Kappa B
- Cardiac-Specific Disruption of Raf-1 Gene Induces Apoptosis and Cardiac Dysfunction
- The Antioxidant N-2-Mercaptopropionyl Glycine Attenuates Left Ventricular Hypertrophy in Murine Pressure-Overloaded Model
- Disruption of a single copy of the p38alpha mitogen-activated protein kinase gene leads to cardioprotection against ischemia-reperfision
- Involvement of ASK1 in G-protein coupled receptor agonistinduced cardiac hypertrophy
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