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東京大学 医系研究 循環器内科 | 論文
- OE-245 Critical Role of Cardiac Angiogenesis in Regulating Cardiac Hypertrophy and Function(Cardiac hypertrophy, basic/clinical-1 (M) OE41,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
- Critical Role of Cardiac Angiogenesis in Regulating Cardiac Hypertrophy and Function(Cardiac Hypertrophy 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- P489 サリン中毒 : 循環器の立場から
- 弁膜疾患 (New 心臓血管外科手術マニュアル) -- (3章 心臓血管外科領域で扱う主な疾患)
- 0117 ずり応力下の内皮細胞における転写因子ETS-1の発現
- OJ-068 Protective Roles of Heat Shock Transcription Factor-1 in Physiological and Pathological Cardiac Hypertrophy(Cardiac hypertrophy, basic/clinical-2 (M) OJ12,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese C
- FRS-171 Role of Na+-Ca2+ Exchanger in Myocardial Ischemia/Reperfusion Injury : Evaluation Using a Heterozygous Na+-Ca2+ Exchanger Knockout Mouse Model(Coronary Heart Disease : Basic Science (IHD) : FRS20)(Featured Research Session (English))
- PJ-577 Stromal Cell-Derived Factor-1 Is Upregulated in the Infarcted Heart and Mediates Migration of Hematopoietic Stem Cells to the Infarcted Heart(Vasoactive Compound/Growth Factor/Cytokine 2 (H) : PJ97)(Poster Session (Japanese))
- OE-008 G-CSF activates Jak/STAT signaling cascade and increases VEGF expression in cardiac myocytes(Vasoactive Compound/Growth Factor/Cytokine 1 (H) : OE2)(Oral Presentation (English))
- Hypertrophic Responses of Cardiomyocytes Induced by Endothelin-1 through the Protein Kinase C-Dependent but Src and Ras-Independent Pathways
- Cardiomyocyte Apoptosis Is a Cause of Heart Failure : A New Mouse Model of Heart Failure
- Angiotensin II Type 1a Receptor Mediates Doxorubicin-Induced Cardiomyopathy
- Stromal Cell-Derived Factor-1 Mediates Migration of Hematopoletic Stem Cells to the Heart after Myocardial Infarction
- Angiotensin II Type 1a Receptor Plays a Critical Role in Cell Infiltration, Cytokine Production and Angiogenesis in Infarcted Myocardium
- Different gene expressions in volume overload and pressure overload hypertrophy
- Junctophilin-2 plays an important role in isoproterenol-induced cardiomyopathy
- Cardioprotective genes are elevated in physiological hypertrophy
- Targeted lacking of ryanodine receptor type 2 in the heart suppresses pressure overload-induced hypertrophic responses but impairs cardiac functions
- Inhibitory Molecules in Signal Transduction Pathways of Cardiac Hypertrophy
- Angiotensin II Induces Hypertrophic Responses through Extracellular Calcium Influx and Activation of CaMKII in Cultured Cardiomyocytes