Sawaki Daigo | Department of Cardiology, Graduate School of Medicine, University of Tokyo

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概要

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著作論文

PE-301 Kruppel-like Factor 6 is a Key Transcription Factor in Adipocyte Differentiation(PE051,Metabolic Syndrome 3 (H),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
PE-260 Positive Regulation of Promoter-Region Specific Histone Incorporation by Histone Chaperones ANP32B and TAF-I, Histone Deacetylase HDAC1 and DNA-Binding Factor KLF5(PE044,Molecular Biology (M),Poster Session (English),The 73rd Annual Scientific Meet
DPE-072 A Novel Pathogenic Pathway of DNA Damage Response as Mediated by KLF5 and Its Transcriptional Complexes in the Cardiovasculature(DPE12,Cardiomyopathy/Hypertrophy (Basic) (M),Digital Poster Session (English),The 73rd Annual Scientific Meeting of Th
DPE-067 Kruppel-like Factor 6 Modulates Cardiovascular Fibrosis(DPE12,Cardiomyopathy/Hypertrophy (Basic) (M),Digital Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
OE-398 Different Molecular Forms of Brain Natriuretic Peptide Measured Using Mass Spectrometry-based Immunoassay are a New Biomarker of Coronary Artery Disease(OE67,Coronary Revascularization, PCI (Restenosis/Others) 3 (IHD),Oral Presentation (English),Th
PE-165 Acyclic Retinoid Inhibits Vascular Remodeling by regulating KLF5(Cardiovascular pharmacology, basic/clinical(03)(H),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OE-258 Kruppel-like Factor 6 is a Key Transcription Factor That Controls Cardiac Fibrosis(Cardiac hypertrophy, basic/clinical(01)(M),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OE-173 A novel pathogenic pathway of DNA damage response as mediated by KLF5 and its transcriptional complexes in the cardiovasculature(Molecular biology, vascular(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circ
OJ-259 Acyclic Retinoid Inhibits Neointima Formation through Retinoic Acid Receptor Beta-induced Apoptosis(Cardiovascular pharmacology, basic/clinical-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-183 Kruppel-like Factor 5 Directly Stimulates Cell Growth in Vascular Lesions(Vascular smooth muscle-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-173 A Novel Regulatory Pathway of DNA Damage Response to Pathologic Vascular Injury as Mediated by KLF5 and its Transcriptional Complexes(Molecular biology, vascular-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-048 Angiotensin II Activates the DNA Damage Response through Nitric Oxide in Vascular Endothelial Cells(Cardiac hypertrophy, basic/clinical-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-009 Modulation of Ubiquitin-proteasomal Degradation of KLF5 Influences Vascular Remodeling(Molecular biology, vascular-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-008 Cellular Proteomic Approach to Identify Transforming Growth Factor (TGF)-beta Induced DNA and/or RNA-binding Factors(Molecular biology, vascular-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-299 Acyclic Retinoid is a Possible Oral Therapeutic Agent against Neointimal Hyperplasia(Cardiovascular pharmacology, basic/clinical-1 (H) OE50,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Socie
OE-023 The Effect of Adenovirus-mediated Overexpression of the Cardiovascular Transcription Factor KLF5 on Vascular Remodeling And Its Mechanism(Molecular biology, vascular-1 (H) OE4,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeti
Acyclic Retinoid Prevents Cardiovascular Remodeling by Regulated KLF5(Cardiovascular Pharmacology, Basic/Clinical 1 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
The Cardiovascular Transcription Factor Kruppel-like Factor 5 is Negatively Regulated by the Deacetylase HDAC1 through Direct Interaction(Molecular Biology, Vascular 1 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Proteomic Identification of a Novel Regulatory Pathway of Pathologic Induction of PDGF-A as Mediated by KLF5 with Transcriptional Cofactors(Molecular Biology, Vascular 1 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PE-267 Impact of Smoking and HDL-C on Coronary Plaque Regression during Statin Therapy in Acute Coronary Syndrome (ACS) : Sub-analysis of JAPAN-ACS(PE045,ACS/AMI (Clinical/Treatment) 2 (IHD),Poster Session (English),The 73rd Annual Scientific Meeting of T
A Case of Heparin-Induced Thrombocytopenia With Sepsis and Congestive Heart Failure : First Autopsy Report in Japan
OE-321 Accumulation of primitive cells in intracoronary thrombi : Temporal analysis of cellular-constituents of the thrombotic materials in patients with myocardial infarction(Acute myocardial infarction, clinical(pathophysiology)(01)(IHD),Oral Presentati
PJ-427 Early Accumulation of Primitive Cells into Intracoronary Thrombi in Patients with Acute Coronary Syndrome(Thromboembolism/Antithrombotic therapy/Thrombolysis-4 (H) PJ72,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the
OE-301 "Black-hole phenomenon" may contribute to positive vascular remodeling with stent malapposition after implantation of sirolimus-eluting stents in swine coronary arteries(Intravascular endoscopy/Intravascular ultrasound(01)(I),Oral Presentation(Engl
Temporal Analysis of Cellular Constituents of Intracoronary Thrombi in ACS Patients Aspirated Through Thrombectomy(Acute Coronary Syndrome, Basic/Clinical 4 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Risk Model of Cardiovascular Surgery in 845 Marfan Patients Using the Japan Adult Cardiovascular Surgery Database
Targeting Transforming Growth Factor-β Signaling in Aortopathies in Marfan Syndrome
Targeting Transforming Growth Factor-β Signaling in Aortopathies in Marfan Syndrome

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