石原 智明 | Institute of Immunological Science, Hokkaido University
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概要
Institute of Immunological Science, Hokkaido University | 論文
- A specific T-cell subset with CD4+/CD38- markers derived from HIV-1 cariers induses apoptosis in healthy donor-derived T-lymphocytes.
- Superoxide generation by monocytes following infection with human cytomegalovirus.
- Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25+ killer cells in a small subset, leading to induction of apoptosis in bystander cells.
- Protease-defective, gp120-containing human immunodeficiency virus type 1 particles induce apoptosis more efficiently than does wild-type virus or recombinant gp120 protein in healthy donor-derived peripheral blood T cells.
- Dependence of host cell cycle for activation of human immunodeficiency virus type 1 gene expression from latency